Ketamine Drug Interactions
Organized by mechanism and clinical effect.
CNS depressants
MechanismSynergistic CNS and respiratory suppression
Clinical effectIncreased sedation, hypoventilation, coma, death risk
- Opioids (morphine, fentanyl, oxycodone)
- Benzodiazepines (midazolam, lorazepam, diazepam)
- Alcohol
- Barbiturates
- Z-hypnotics (zolpidem)
- Gabapentinoids (gabapentin, pregabalin)
- Other sedatives / anesthetics (propofol)
Sympathomimetic agents
MechanismKetamine increases catecholamines and causes excessive cardiovascular stimulation
Clinical effectSevere hypertension, tachycardia, arrhythmias
- Amphetamines
- Cocaine
- Methylphenidate
- Ephedrine
- Pseudoephedrine
MAO inhibitors (MAOIs)
MechanismExcess catecholamine accumulation and causes hypertensive crisis.
Clinical effectHypertensive crisis, arrhythmias, hyperthermia
- Phenelzine
- Tranylcypromine
- Isocarboxazid
- Linezolid (functional MAOI)
Antidepressants (SSRIs, SNRIs, TCAs)
SSRIs / SNRIs
- Fluoxetine, sertraline, venlafaxine, duloxetine
Effect Usually safe; possible increased BP/HR; rare serotonin toxicity (especially with high doses or polypharmacy).
TCAs
- Amitriptyline, imipramine
Effect Increased arrhythmia risk; additive anticholinergic and CNS effects.
Antipsychotics
MechanismDopamine antagonism vs ketamine psychotomimetic effects
Clinical effectVariable sedation; QT risk
- Haloperidol → decreases emergence reactions
- Atypicals (quetiapine, risperidone)
CYP450 interactions (metabolism effects)
MetabolismKetamine is metabolized mainly by CYP3A4, CYP2B6, and CYP2C9.
CYP inhibitors cause increased ketamine levels
- Clarithromycin
- Erythromycin
- Azoles (ketoconazole)
- Protease inhibitors
- Grapefruit juice
EffectProlonged dissociation, sedation, psychosis.
CYP inducers cause decreased ketamine levels
- Rifampin
- Carbamazepine
- Phenytoin
- Phenobarbital
- St. John’s wort
EffectReduced anesthetic/antidepressant efficacy.
Antihypertensives
MechanismOpposing hemodynamic effects
Clinical effectHypotension or unpredictable BP swings
- Beta-blockers
- ACE inhibitors
- ARBs
- Calcium channel blockers
Theophylline
MechanismLowers seizure threshold
Clinical effectSeizures
- Theophylline
- Aminophylline
Anticholinergic agents
MechanismAdditive antimuscarinic effects
Clinical effectDelirium, agitation, tachycardia
- Atropine
- Scopolamine
- TCAs
- First-generation antihistamines
Other notable interactions
- Lithium → increased neurotoxicity, confusion
- Levodopa → exaggerated BP/HR response
- Neuromuscular blockers causes prolonged weakness
- Valproate causes increased sedation